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- W4212977396 abstract "Epicardial adipose tissue surrounds and infiltrates the heart. Epicardial fat displays unique anatomic, genetic, and biomolecular properties. People with obesity and in particular, those with abdominal obesity and associated type 2 diabetes mellitus, have an increased amount of epicardial adipose tissue (EAT). Epicardial fat works well as therapeutic target due to its fast-responding metabolism, organ fat specificity, and easy measurability. Epicardial fat responds to thiazolidinediones (TZD), glucagon-like peptide 1-receptor agonists (GLP1A), sodium-glucose cotransporter 2 inhibitors (SGLT2i), dipeptidyl peptidase-4 inhibitors (DPP4i), and statins. Modulating epicardial fat morphology and genetic profile with targeted pharmacological agents suggests novel strategies in the pharmacotherapy of diabetes and obesity." @default.
- W4212977396 created "2022-02-24" @default.
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- W4212977396 creator A5047034112 @default.
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- W4212977396 date "2022-01-01" @default.
- W4212977396 modified "2023-09-25" @default.
- W4212977396 title "Targeting Epicardial Fat in Obesity and Diabetes Pharmacotherapy" @default.
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- W4212977396 doi "https://doi.org/10.1007/164_2021_577" @default.
- W4212977396 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/35156138" @default.
- W4212977396 hasPublicationYear "2022" @default.
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