Matches in SemOpenAlex for { <https://semopenalex.org/work/W4214853881> ?p ?o ?g. }
- W4214853881 abstract "Gut dysbiosis is defined as disorders of gut microbiota and loss of barrier integrity, which are ubiquitous on pathological conditions and associated with the development of various diseases. Kidney diseases are accompanied with gut dysbiosis and metabolic disorders, which in turn contribute to the pathogenesis and progression of kidney diseases. Microbial alterations trigger production of harmful metabolites such as uremic toxins and a decrease in the number of beneficial ones such as SCFAs, which is the major mechanism of gut dysbiosis on kidney diseases according to current studies. In addition, the activation of immune responses and mitochondrial dysfunction by gut dysbiosis, also lead to the development of kidney diseases. Based on the molecular mechanisms, modification of gut dysbiosis via probiotics, prebiotics and synbiotics is a potential approach to slow kidney disease progression. Fecal microbiota transplantation (FMT) and genetic manipulation of the gut microbiota are also promising choices. However, the clinical use of probiotics in kidney disease is not supported by the current clinical evidence. Further studies are necessary to explore the causal relationships of gut dysbiosis and kidney diseases, the efficiency and safety of therapeutic strategies targeting gut-kidney axis." @default.
- W4214853881 created "2022-03-05" @default.
- W4214853881 creator A5033056656 @default.
- W4214853881 creator A5045460123 @default.
- W4214853881 creator A5061908394 @default.
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- W4214853881 date "2022-03-03" @default.
- W4214853881 modified "2023-10-17" @default.
- W4214853881 title "Gut Dysbiosis and Kidney Diseases" @default.
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- W4214853881 doi "https://doi.org/10.3389/fmed.2022.829349" @default.
- W4214853881 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/35308555" @default.
- W4214853881 hasPublicationYear "2022" @default.
- W4214853881 type Work @default.