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- W4220845226 endingPage "3315" @default.
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- W4220845226 abstract "One of the major mechanisms of drug-induced liver injury includes mitochondrial perturbation and dysfunction. This is not a surprise, given that mitochondria are essential organelles in most cells, which are responsible for energy homeostasis and the regulation of cellular metabolism. Drug-induced mitochondrial dysfunction can be influenced by various factors and conditions, such as genetic predisposition, the presence of metabolic disorders and obesity, viral infections, as well as drugs. Despite the fact that many methods have been developed for studying mitochondrial function, there is still a need for advanced and integrative models and approaches more closely resembling liver physiology, which would take into account predisposing factors. This could reduce the costs of drug development by the early prediction of potential mitochondrial toxicity during pre-clinical tests and, especially, prevent serious complications observed in clinical settings." @default.
- W4220845226 created "2022-04-03" @default.
- W4220845226 creator A5018211217 @default.
- W4220845226 creator A5045862213 @default.
- W4220845226 date "2022-03-18" @default.
- W4220845226 modified "2023-09-30" @default.
- W4220845226 title "Mitochondria as the Target of Hepatotoxicity and Drug-Induced Liver Injury: Molecular Mechanisms and Detection Methods" @default.
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