FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W4220933664> ?p ?o ?g. }

• W4220933664 endingPage "1723" @default.
• W4220933664 startingPage "1722" @default.
• W4220933664 abstract "Journal of Cellular and Molecular MedicineVolume 26, Issue 5 p. 1722-1723 CORRIGENDUMOpen Access This article corrects the following: LKB1IP promotes pathological cardiac hypertrophy by targeting PTEN/Akt signalling pathway Mi Tian, Xiuxin Jiang, Xinyun Li, Jianmin Yang, Cheng Zhang, Wencheng Zhang, Volume 25Issue 5Journal of Cellular and Molecular Medicine pages: 2517-2529 First Published online: January 24, 2021 First published: 06 March 2022 https://doi.org/10.1111/jcmm.17194AboutSectionsPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinked InRedditWechat In Tian M et al.,1 the published article contains errors in Figure 3D and Figure 6D. The correct figures are shown below. The authors confirm all results and conclusions of this article remain unchanged. FIGURE 1Open in figure viewerPowerPoint LKB1IP deficiency alleviates ISO-induced cardiac hypertrophy. (A) Representative M-mode echocardiography of the left ventricle (top). Measurement of diastolic interventricular septal thickness (IVSd), systolic interventricular septal thickness (IVSs), diastolic left ventricular posterior wall (LVPWd) and systolic left ventricular posterior wall (LVPWs) in the indicated groups (n = 5). *p < 0.05, **p < 0.01 ***p < 0.001 vs. WT Saline; # p < 0.05, ## p < 0.01 vs. WT ISO. (B) Quantitative analysis of ratio of heart weight to body weight (HW/BW) in the indicated groups (n = 5). **p < 0.01 vs. WT Saline; # p < 0.05 vs. WT ISO. (C) Quantitative analysis of ratio of HW to tibial length (TL) in the indicated groups (n=5). *p < 0.05, **p < 0.05 vs. WT Saline; # p < 0.05 vs. WT ISO. (D) Analysis of whole hearts (the first row; scale bar, 1000 μm) and heart sections stained with wheat germ agglutinin (WGA; the second row; scale bar, 50 μm) or picosirius red (PSR; the third and fourth rows; scale bars, 50 μm) from the indicated groups 7 days after saline or ISO treatment. (E) Quantitative analysis of the average cardiomyocyte cross-sectional area in the indicated groups, n > 100 cells per group. **p < 0.01, ***p < 0.001 vs. WT Saline; ## p < 0.01 vs. WT ISO. (F) Quantitative analysis of left ventricle (LV) collagen volume in the indicated groups, n > 15 fields per group. **p < 0.01, *** p < 0.001 vs. WT Saline; ### p < 0.001 vs. WT ISO. (G) Quantitative PCR analysis of ANP, BNP and β-MHC mRNA levels in the indicated groups (n = 3). *p < 0.05 vs. WT Saline; # p < 0.05 vs. WT ISO FIGURE 2Open in figure viewerPowerPoint LKB1IP positively regulates Akt phosphorylation. (A) Western blot analysis of phosphorylated Akt from hearts of ISO-infused WT and LKB1IP-/- mice (n = 4). *p < 0.05, **p < 0.01 vs. WT Saline; # p < 0.05 vs. WT ISO. (B) Western blot analysis of phosphorylated Akt level in hearts of WT and LKB1IP-/- mice after TAC surgery (n = 4). **p < 0.01, ***p < 0.001 vs. WT Sham; #### p < 0.0001 vs. WT TAC. (C) Western blot analysis of phosphorylated Akt level in NRCMs infected with AdGFP or AdLKB1IP followed by ISO treatment (n = 3). ***p < 0.001 vs. AdGFP Saline; ## p < 0.01 vs. AdGFP ISO. (D) Western blot analysis of phosphorylated Akt level in NRCMs transfected with control or LKB1IP siRNA followed by ISO treatment (n = 3). **p < 0.01, ***p < 0.001 vs. Control siRNA Saline; ## p < 0.01 vs. Control siRNA ISO REFERENCE 1 Tian M, Jiang X, Li X, Yang J, Zhang C, Zhang W. LKB1IP promotes pathological cardiac hypertrophy by targeting PTEN/Akt signalling pathway. J Cell Mol Med. 2021; 25(5): 2517– 2529. 10.1111/jcmm.16199. Wiley Online LibraryCASPubMedWeb of Science®Google Scholar Volume26, Issue5March 2022Pages 1722-1723 FiguresReferencesRelatedInformation" @default.
• W4220933664 created "2022-04-03" @default.
• W4220933664 date "2022-03-01" @default.
• W4220933664 modified "2023-10-17" @default.
• W4220933664 title " " @default.
• W4220933664 cites W3121493982 @default.
• W4220933664 doi "https://doi.org/10.1111/jcmm.17194" @default.
• W4220933664 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/35253382" @default.
• W4220933664 hasPublicationYear "2022" @default.
• W4220933664 type Work @default.
• W4220933664 citedByCount "0" @default.
• W4220933664 crossrefType "journal-article" @default.
• W4220933664 hasBestOaLocation W42209336641 @default.
• W4220933664 hasConcept C126322002 @default.
• W4220933664 hasConcept C164705383 @default.
• W4220933664 hasConcept C166957645 @default.
• W4220933664 hasConcept C167414201 @default.
• W4220933664 hasConcept C185592680 @default.
• W4220933664 hasConcept C190283241 @default.
• W4220933664 hasConcept C191935318 @default.
• W4220933664 hasConcept C2777045944 @default.
• W4220933664 hasConcept C2777609662 @default.
• W4220933664 hasConcept C2778921608 @default.
• W4220933664 hasConcept C55493867 @default.
• W4220933664 hasConcept C57900726 @default.
• W4220933664 hasConcept C71924100 @default.
• W4220933664 hasConcept C84393581 @default.
• W4220933664 hasConcept C86554907 @default.
• W4220933664 hasConcept C95457728 @default.
• W4220933664 hasConceptScore W4220933664C126322002 @default.
• W4220933664 hasConceptScore W4220933664C164705383 @default.
• W4220933664 hasConceptScore W4220933664C166957645 @default.
• W4220933664 hasConceptScore W4220933664C167414201 @default.
• W4220933664 hasConceptScore W4220933664C185592680 @default.
• W4220933664 hasConceptScore W4220933664C190283241 @default.
• W4220933664 hasConceptScore W4220933664C191935318 @default.
• W4220933664 hasConceptScore W4220933664C2777045944 @default.
• W4220933664 hasConceptScore W4220933664C2777609662 @default.
• W4220933664 hasConceptScore W4220933664C2778921608 @default.
• W4220933664 hasConceptScore W4220933664C55493867 @default.
• W4220933664 hasConceptScore W4220933664C57900726 @default.
• W4220933664 hasConceptScore W4220933664C71924100 @default.
• W4220933664 hasConceptScore W4220933664C84393581 @default.
• W4220933664 hasConceptScore W4220933664C86554907 @default.
• W4220933664 hasConceptScore W4220933664C95457728 @default.
• W4220933664 hasIssue "5" @default.
• W4220933664 hasLocation W42209336641 @default.
• W4220933664 hasLocation W42209336642 @default.
• W4220933664 hasLocation W42209336643 @default.
• W4220933664 hasOpenAccess W4220933664 @default.
• W4220933664 hasPrimaryLocation W42209336641 @default.
• W4220933664 hasRelatedWork W1600397528 @default.
• W4220933664 hasRelatedWork W1976602181 @default.
• W4220933664 hasRelatedWork W2365061539 @default.
• W4220933664 hasRelatedWork W2392611692 @default.
• W4220933664 hasRelatedWork W2408907719 @default.
• W4220933664 hasRelatedWork W2411389335 @default.
• W4220933664 hasRelatedWork W2417271704 @default.
• W4220933664 hasRelatedWork W2431765988 @default.
• W4220933664 hasRelatedWork W3201089159 @default.
• W4220933664 hasRelatedWork W3206615596 @default.
• W4220933664 hasVolume "26" @default.
• W4220933664 isParatext "false" @default.
• W4220933664 isRetracted "false" @default.
• W4220933664 workType "article" @default.