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- W4220956391 abstract "The increasing number of patients with chronic kidney disease (CKD) is being recognized as an emerging global health problem. Recently, it has become clear that injury and loss of glomerular visceral epithelial cells, known as podocytes, is a common early event in many forms of CKD. Podocytes are highly specialized epithelial cells that cover the outer layer of the glomerular basement membrane. They serve as the final barrier to urinary protein loss through the formation and maintenance of specialized foot-processes and an interposed slit-diaphragm. We previously reported that the transcription factor MafB regulates the podocyte slit diaphragm protein production and transcription factor Tcf21. We showed that the forced expression of MafB was able to prevent CKD. In this review, we discuss recent advances and offer an updated overview of the functions of podocyte-specific transcription factors in kidney biology, aiming to present new perspectives on the progression of CKD and respective therapeutic strategies." @default.
- W4220956391 created "2022-04-03" @default.
- W4220956391 creator A5000730705 @default.
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- W4220956391 creator A5079821787 @default.
- W4220956391 date "2023-01-01" @default.
- W4220956391 modified "2023-09-25" @default.
- W4220956391 title "Podocyte-specific Transcription Factors: Could MafB Become a Therapeutic Target for Kidney Disease?" @default.
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- W4220956391 doi "https://doi.org/10.2169/internalmedicine.9336-22" @default.
- W4220956391 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/35249929" @default.
- W4220956391 hasPublicationYear "2023" @default.
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