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• W4220960281 abstract "Immunoglobulin A (IgA) vasculitis (IgAV), also known as Henoch-Schönlein purpura, is the most common form of childhood vasculitis. It is characterized by cutaneous hemorrhage, resulting from red blood cell leakage into the skin or mucosae, possibly caused by damage to small blood vessels. These acute symptoms usually disappear without treatment. Endothelial cells are distributed on the inner surfaces of blood vessels and lymphatic vessels, and have important functions in metabolism and endocrine function, as well as being the primary targets of external stimuli and endogenous immune activity. Injury to endothelial cells is a feature of IgA vasculitis. Endothelial cell damage may be related to the deposition of immune complexes, the activation of complement, inflammatory factors, and chemokines, oxidative stress, hemodynamics, and coagulation factors. Both epigenetic mechanisms and genetic diversity provide a genetic background for endothelial cell injury. Here, research on the role of endothelial cells in allergic IgA vasculitis is reviewed." @default.
• W4220960281 created "2022-04-03" @default.
• W4220960281 creator A5020528044 @default.
• W4220960281 creator A5042570642 @default.
• W4220960281 creator A5052180002 @default.
• W4220960281 creator A5058715571 @default.
• W4220960281 creator A5080932283 @default.
• W4220960281 creator A5090875147 @default.
• W4220960281 date "2022-03-16" @default.
• W4220960281 modified "2023-09-26" @default.
• W4220960281 title "A Review of the Mechanism of Vascular Endothelial Injury in Immunoglobulin A Vasculitis" @default.
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