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- W4220966154 abstract "Multiple sclerosis (MS) is a neuroinflammatory disease characterized by immune cell infiltration in the central nervous system and destruction of myelin sheaths. Alterations of gut bacteria abundances are present in MS patients. In mouse models of neuroinflammation, depletion of microbiota results in amelioration of symptoms, and gavage with MS patient microbiota exacerbates the disease and inflammation via Th17 cells. On the other hand, depletion of B cells using anti-CD20 is an efficient therapy in MS, and growing evidence shows an important deleterious role of B cells in MS pathology. However, the failure of TACI-Ig treatment in MS highlighted the potential regulatory role of plasma cells. The mechanism was recently demonstrated involving IgA+ plasma cells, specific for gut microbiota and producing IL-10. IgA-coated bacteria in MS patient gut exhibit also modifications. We will focus our review on IgA interactions with gut microbiota and IgA+ B cells in MS. These recent data emphasize new pathways of neuroinflammation regulation in MS." @default.
- W4220966154 created "2022-04-03" @default.
- W4220966154 creator A5027556492 @default.
- W4220966154 creator A5055337829 @default.
- W4220966154 creator A5059903212 @default.
- W4220966154 date "2022-03-14" @default.
- W4220966154 modified "2023-10-16" @default.
- W4220966154 title "Microbiota, IgA and Multiple Sclerosis" @default.
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