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- W4221127711 abstract "Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), the causative agent of coronavirus disease 2019 (COVID-19), is spreading throughout the world at an alarming rate.[1] Severe disease is characterized by acute respiratory distress syndrome (ARDS), frequently associated with thrombotic complications.[2] [3] Some studies report the presence of platelets in thrombi found in multiple organs of COVID-19 autopsy cases.[4] [5] Moreover, hyperreactive platelets and overwhelming generation of inflammatory cytokines (i.e., a condition also known as “cytokine storm”) are frequent in COVID-19 patients.[6] [7] [8] [9] [10] [11] Furthermore, SARS-CoV-2 ribonucleic acid (RNA) could be detected in platelets of COVID-19 patients.[6] [10] [12] Despite these findings suggesting a role for platelets in the pathophysiology of COVID-19, the mechanisms of increased thrombotic events remain not completely elucidated." @default.
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- W4221127711 date "2022-03-07" @default.
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- W4221127711 title "Aspirin use Reduces Platelet Hyperreactivity and Degranulation in COVID-19 Patients" @default.
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- W4221127711 doi "https://doi.org/10.1055/s-0042-1744281" @default.
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