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• W4225133059 abstract "Postsurgical thrombotic microangiopathy (TMA) is a complication associated with significant morbidity and mortality. Still, the pathophysiological underlying mechanism of postsurgical TMA, a diagnosis often overlooked in postoperative patients with acute kidney injury and thrombocytopenia, is largely unknown. Here, we report the case of a 56-year-old male that developed anuric acute kidney injury, Coombs-negative hemolysis, and thrombocytopenia after surgical aortic arch replacement. Massive ex vivo complement activation on the endothelium, a rare complement gene variant in C2, at-risk haplotype MCPggaac, and excellent response to therapeutic complement inhibition, points to the pivotal role of complement in the pathophysiology of disease. Moreover, the importance of a multidisciplinary team approach in (postsurgical) thrombocytopenia is emphasized." @default.
• W4225133059 created "2022-05-01" @default.
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• W4225133059 date "2022-04-29" @default.
• W4225133059 modified "2023-09-24" @default.
• W4225133059 title "Postsurgical Thrombotic Microangiopathy and Deregulated Complement" @default.
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• W4225133059 doi "https://doi.org/10.3390/jcm11092501" @default.
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