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- W4229748838 abstract "RETRACTIONRetractionPublished Online:01 Sep 2005https://doi.org/10.1152/ajpheart.01246.2004This is the final version - click for previous versionMoreSectionsPDF (22 KB)Download PDF ToolsExport citationAdd to favoritesGet permissionsTrack citations ShareShare onFacebookTwitterLinkedInEmail April 8, 2005doi:10.1152/ajpheart.01246.2004: Islamovic, Emir, William T. Gerthoffer, and Ruben Mestril. “Hsp20 is essential for the viability of neonatal rat cardiomyocytes.” The authors recently discovered that part of the published results were invalid because some of the studies were conducted using adenoviral vectors containing HSP20 constructs different from those they thought they had used. That is, in one study the vector used contained a fragment instead of the full-length HSP20 in the sense orientation. In another study, instead of using vectors containing a full-length, non-phosphorylatable HSP20 mutant, the authors used one containing a fragment of HSP20 lacking the phosphorylation mutation. Although the experiments conducted using these two constructs do not have impact on the main conclusion of the study, viz., HSP20 is essential for myocyte viability, all the authors believe it is improper to proceed with final publication of this manuscript in its present form. Therefore, this article is being retracted by the publisher, the American Physiological Society, at the request of the authors. The authors deeply regret this error and apologize for any inconvenience that the publication of this study may have caused.This article has no references to display. Previous Back to Top FiguresReferencesRelatedInformation More from this issue > Volume 289Issue 3September 2005Pages H1321-H1321 Copyright & PermissionsCopyright © 2005 by the American Physiological Societyhttps://doi.org/10.1152/ajpheart.01246.2004History Published online 1 September 2005 Published in print 1 September 2005 PDF download Metrics Downloaded 76 times" @default.
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