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- W42298543 abstract "In the classical endocrine renin—angiotensin system (RAS), renin secreted from the kidney cleaves hepatic angiotensinogen in the circulation to launch a cascade of angiotensin peptides including angiotensin (A) I, II, and III. AII, the most prominent of these peptide hormones, initiates through specific plasma membrane receptors, a host of physiological actions ranging from vasoconstriction to volume and growth regulation (1-4). Circulating plasma levels of renin and/or AII do not always correlate with their purported functions (5, 6),because of inaccuracies of renin and/or AII measurements, or not considering the sodium status (7). Studies with angiotensin-converting enzyme (ACE) inhibitors, and more recently with specific AII receptor antagonists, have demonstrated that even when the circulating levels are low, AII remains a major regulator of many biological responses, including blood pressure (8)." @default.
- W42298543 created "2016-06-24" @default.
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- W42298543 date "1996-01-01" @default.
- W42298543 modified "2023-09-24" @default.
- W42298543 title "The Vascular Renin System" @default.
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- W42298543 doi "https://doi.org/10.1007/978-1-4612-0231-8_15" @default.
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