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- W4234831885 abstract "Injurious mechanical, thermal, or chemical stimuli acting on somatic and visceral tissues are encoded into a discharge of nerve impulses by peripheral endings of Aδ and C nociceptor neurons located at sensory ganglia. Nociceptors are functionally classified as mechano-, polymodal, and silent nociceptors. Noxious stimuli are transduced by membrane receptor/channel proteins at nociceptor endings into depolarizing currents that evoke propagated nerve impulses into their parent axon. Inflammatory mediators, released by tissue injury, act on membrane receptors through second messenger cascades, causing decreased threshold and enhanced spontaneous and stimulus-evoked impulse activity (sensitization). Damage to peripheral nociceptor neurons also changes membrane protein expression and causes long-lasting disturbances in excitability and impulse firing." @default.
- W4234831885 created "2022-05-12" @default.
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- W4234831885 date "2009-01-01" @default.
- W4234831885 modified "2023-09-26" @default.
- W4234831885 title "Nociceptor Responses" @default.
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- W4234831885 doi "https://doi.org/10.1016/b978-008045046-9.01925-2" @default.
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