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- W4236098974 abstract "We have uncovered an error in the C-terminal sequence of the expressed telencephalin (tln) cDNA that we generated for expression in hippocampal neurons (J. N. Stowell and A. M. Craig [1999]. Neuron 22, 525–536). Replication of the experiments with the correct full-length clone showed that, unlike endogenous tln (1Benson D.L. Yoshihara Y. Mori K. Polarized distribution and cell type–specific localization of telencephalin, an intercellular adhesion molecule.J. Neurosci. Res. 1998; 52: 43-53Crossref PubMed Scopus (38) Google Scholar), rabbit tln expressed from the HSV vector was not excluded from axons in most neurons in our cultures. The addition of the mGluR7 C terminus to full-length tln (construct tln–tail7) did increase targeting to axons, particularly at early times after transfection, but the difference was less dramatic than that shown in Figure 6 (the figure below presents the data with the correct clone). This slighter difference and the ability of the mGluR7 C terminus to redirect full-length mGluR2 to axons still support our hypothesis that the mGluR7 C terminus functions as a dominant axon targeting signal. We apologize to our colleagues for any confusion caused by this error." @default.
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- W4236098974 date "1999-11-01" @default.
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- W4236098974 title "Axon/Dendrite Targeting of Metabotropic Glutamate Receptors by Their Cytoplasmic Carboxy-Terminal Domains" @default.
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- W4236098974 doi "https://doi.org/10.1016/s0896-6273(00)81128-9" @default.
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