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- W4238234488 endingPage "320" @default.
- W4238234488 startingPage "311" @default.
- W4238234488 abstract "Immune status is altered in patients with sepsis or non-infectious systemic inflammatory response syndrome (SIRS). Reduced ex-vivo TNF production by endotoxin-activated monocytes has been regularly reported. This observation is reminiscent of the phenomenon of endotoxin tolerance, and the term `leukocyte reprogramming' well defines this phenomenon. This review will outline that the hyporesponsiveness of circulating leukocytes is not a generalized phenomenon in sepsis and SIRS. Indeed, the nature of the insult ( i.e. infectious versus non-infectious SIRS; under anesthesia [surgery] or not [trauma, burn]), the nature of the activator used to trigger leukocytes ( i.e. different Toll-like receptor ligands or whole bacteria), the nature of the cell culture ( i.e. isolated monocytes versus peripheral blood mononuclear cells versus whole blood assays), and the nature of the analyzed cytokines ( e.g. IL-1β versus IL-1ra; TNF versus IL-10) have a profound influence on the outcome of the response." @default.
- W4238234488 created "2022-05-12" @default.
- W4238234488 creator A5036995113 @default.
- W4238234488 creator A5057372078 @default.
- W4238234488 creator A5063108232 @default.
- W4238234488 creator A5072038022 @default.
- W4238234488 date "2005-10-01" @default.
- W4238234488 modified "2023-09-30" @default.
- W4238234488 title "Reprogramming of circulatory cells in sepsis and SIRS" @default.
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