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- W4242309199 abstract "These correspondents suggest that we were intemperate in considering that the amount of oxalic acid excreted after the intravenous infusion of approximately 100 g ascorbic acid may create only a moderate risk in people with normal renal function. Whether the risk of oxalate stone formation (or acute nephropathy) from high-dose intravenous ascorbic acid is small, moderate, or large in a given situation is a clinical judgment that most physicians would balance against the benefit of the treatment. Undoubtedly, intravenous vitamin C is an unproven treatment. Our report is useful because it describes a method for overcoming the technical problem of analyzing oxalic acid in the presence of extremely high ascorbic acid concentrations and because of the surprisingly small fractional conversion of ascorbic acid to oxalic acid under the conditions of our study. Intravenous ascorbic acid infusions and oxalate productionMetabolism - Clinical and ExperimentalVol. 58Issue 6PreviewRobitaille et al [1] studied the extent of the autooxidation of ascorbate to oxalate after the intravenous administration of megadoses of ascorbic acid to patients with advanced cancers of a variety of types. They found that when 100 g of ascorbate was delivered over 1.5 to 2 hours, urinary oxalate excretion reached an average of 81 mg over a 6-hour period. A portion of this oxalate increase was due to the 26 mg present in the infusate. They concluded that this increase in oxalate excretion would not be clinically significant. Full-Text PDF" @default.
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- W4242309199 date "2009-06-01" @default.
- W4242309199 modified "2023-09-27" @default.
- W4242309199 title "Reply: Oxalic acid excretion after intravenous ascorbic acid administration" @default.
- W4242309199 doi "https://doi.org/10.1016/j.metabol.2009.02.007" @default.
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