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- W4246263175 abstract "It is one of the most important achievements in modern basic cardiology that many substances with a positive or negative inotropic effect on heart muscle act as promoters or inhibitors of the mediator function of Ca2+ions in excitation-contraction coupling. For instance, beta adrenergic stimulation with adrenaline, noradrenaline, or isoproterenol facilitates the transmembrane Ca2+influx during excitation. Splitting of ATP by the Ca2+-activated myofibrillar ATPase, and contractile tension development, are thereby augmented. Similarly, under the influence of cardiac glycosides, more Ca2+is made available to the contractile myofibrils. Conversely, a number of negative inotropic substances can inhibit excitation contraction coupling of heart muscle by a Ca2+-antagonistic effect.KeywordsCalcium AntagonistPapillary MuscleTyrode SolutionSlow ChannelMyofibrillar ATPaseThese keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves." @default.
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- W4246263175 date "1989-01-01" @default.
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- W4246263175 title "Effects of and the Mechanism of Action of Calcium Antagonists and Other Antianginal Agents" @default.
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- W4246263175 doi "https://doi.org/10.1007/978-1-4613-0873-7_22" @default.
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