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- W4247291278 abstract "0006 Chronic systemic inflammation, as evidenced by higher than normal cytokines (interleukin-6, tumor necrosis factor alpha; IL-6, TNFα) and the acute-phase reactant C-reactive protein (CRP), is an independent predictor of several chronic diseases, including coronary heart disease, stroke, and diabetes. As an endocrine organ, adipose tissue produces and secretes a number of inflammatory cytokines. There is strong evidence that high concentrations of inflammatory markers are associated with total, and especially abdominal, adiposity. Lifestyle modifications, such as weight loss and exercise, have been advocated for the treatment of obesity and the metabolic syndrome. Several studies now show that weight loss via caloric restriction reduces concentrations of inflammatory markers in obese individuals. While it is well established that strenuous exercise acutely increases levels of circulating cytokines and CRP, recent evidence indicates that long-term exercise training may reduce chronic inflammation. In observational studies, lower levels of several inflammatory markers, are found in individuals who are the most physically active and the most physically fit. Moreover, some intervention studies show that chronic exercise training reduces circulating concentrations of cytokines or their soluble receptors. Addition of exercise training to caloric restriction is more effective than weight loss alone in reducing chronic inflammation in obese individuals. In addition, modification of chronic inflammation is associated with changes in abdominal, but not gluteal, adipose tissue lipolysis in response to weight loss and exercise, suggesting that regional fat metabolism may also be related to chronic inflammation in obese individuals." @default.
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- W4247291278 date "2004-05-01" @default.
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- W4247291278 title "Inflammation and the Metabolic Syndrome" @default.
- W4247291278 doi "https://doi.org/10.1249/00005768-200405001-00003" @default.
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