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• W4247678840 abstract "(Cancer Cell 9, 209–223; March 2006) In this paper, certain images in Figure 3C, Figure 5B, and Figure 6E were mistakenly used during preparation and assembly of the figures; the corrected figures are shown below. The expression of CNTN-1 and β-actin shown in the left middle panel of the original Figure 3C (H928/vector and H928/VEGF-C cells stably transfected with CNTN-1 siRNA2) were not the appropriate figures and should be replaced with the corrected Figure 3 here. Similarly, the expression levels of CNTN-1 and β-actin displayed in the original Figure 6E were incorrect and mistakenly used. The correct image is shown in the new Figure 6. We also regret that several inadvertent duplications took place involving the original Figure 5B and Figure 6D, as some of these images represented similar experiments analyzing primary tumors and lung metastases, respectively, using the same cell lines. This error occurred while submitting the revised manuscript, and the panels displaying immunoblots of tyrosine-phosphorylated Flt-4, expression of β-actin, and expression of VEGF-C were inappropriately replaced with images in Figure 6D. We have replaced all of the images in Figure 5B, which are shown correctly in new Figure 5. All of the corrected data are consistent with our previous conclusions; hence, the experimental results and conclusions of this article are not affected by these modifications. The authors apologize for the inconvenience caused by these errors.Figure 5View Large Image Figure ViewerDownload Hi-res image Download (PPT)Figure 6View Large Image Figure ViewerDownload Hi-res image Download (PPT) The VEGF-C/Flt-4 axis promotes invasion and metastasis of cancer cellsSu et al.Cancer CellMarch, 2006In BriefFlt-4, a VEGF receptor, is activated by its specific ligand, VEGF-C. The resultant signaling pathway promotes angiogenesis and/or lymphangiogenesis. This report provides evidence that the VEGF-C/Flt-4 axis enhances cancer cell mobility and invasiveness and contributes to the promotion of cancer cell metastasis. VEGF-C/Flt-4-mediated invasion and metastasis of cancer cells were found to require upregulation of the neural cell adhesion molecule contactin-1 through activation of the Src-p38 MAPK-C/EBP-dependent pathway. Full-Text PDF Open Archive" @default.
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• W4247678840 date "2008-09-01" @default.
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• W4247678840 title "The VEGF-C/Flt-4 Axis Promotes Invasion and Metastasis of Cancer Cells" @default.
• W4247678840 doi "https://doi.org/10.1016/j.ccr.2008.08.008" @default.
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