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• W4251087131 abstract "We are interested in the study made by Friedman et al. (Friedman SA, Schiff E, Kao L, Sibai BM. Neonatal outcome after preterm delivery for preeclampsia. AM J OBSTET GYNECOL 1995;172:1785-92). This article could not support the idea that fetuses of preeclamptic women have accelerated maturation and improved neonatal outcome because of intrauterine “stress.” We also studied the neonatal morbidity of infants delivered from severely preeclamptic pregnancies. Our findings suggest a conclusion similar to that established by Friedman et al. In our study the neonatal outcome was compared between premature infants born by cesarean section to 52 severe preeclamptic and 30 normotensive women.1Beke A Rigó Jr, J Paulin F The effect of preeclampsia on neonatal morbidity (in Hungarian).Orv Hetil. 1995; 136: 1999-2003PubMed Google Scholar Criteria of severe preeclampsia were defined according to The American College of Obstetricians and Gynecologists (1986). Intrauterine growth retardation was diagnosed if birth weight was <10th percentile for gestational age. Multiple pregnancy, abruptio placentae, intrauterine infection, and diabetes were excluded from the study. The gestational age at delivery was similar in the two groups (preeclamptic 33.0 ± 2.2 weeks vs normotensive 32.3 ± 2.4 weeks). Significant differences were established between the two groups in birth weight (preeclamptic 1501.3 ± 373.3 gm vs normotensive 1916.0 ± 333.3 gm, p < 0.0002) and in the frequency of intrauterine growth retardation (preeclamptic 25/52 vs normotensive 0/30, p < 0.0001). In spite of this, there were no significant differences in the incidence of the following neonatal complications and diseases between the two groups: respiratory distress syndrome, wet lung, pneumothorax, bronchopulmonary dysplasia, patent ductus arteriosus, intraventricular hemorrhage, and hyperviscosity syndrome. However, the average nursing time in the neonatal intensive care unit was significantly longer in the preeclamptic group (preeclamptic 14.6 ± 8.5 days vs normotensive 11.1 ± 5.1 days, p < 0.04). Concerning the laboratory findings, only the average platelet count of the infants differed significantly between the two groups (preeclamptic 211.4 ± 83.4 × 109/L vs normotensive 251.1 ± 88.8 × 109/L, p < 0.04). The incidence of thrombopenic infants (platelet count <100 × 109/L) differed more significantly (preeclamptic 12/52 vs normotensive 1/30, p < 0.01). There was no relationship between the cases of intraventricular hemorrhage and low platelet count. Some data suggest that hydralazine medication of preeclamptic women could induce thrombocytopenia in newborns.2Widerlov E Karlman I Storsater J Hydralazine-induced neonatal thrombocytopenia.N Engl J Med. 1980; 303: 1235PubMed Google Scholar In conclusion, findings by Friedman et al. seem to be in agreement with our study, but it is also true that the neonatal results are not poorer in newborns of preeclamptic women in spite of the lower mean birth weight and the higher incidence of intrauterine growth retardation of this group. 6/8/71500" @default.
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• W4251087131 date "1996-03-01" @default.
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• W4251087131 title "Neonatal outcome after preterm delivery for preeclampsia" @default.
• W4251087131 doi "https://doi.org/10.1016/s0002-9378(96)70362-9" @default.
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