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- W4252410855 abstract "Follistatin acts as an activin-binding protein, which prevents the access of activin to its receptor. In Xenopus, overexpression of follistatin can act as a neural inducer and it has been proposed that this effect may be due to the ability of follistatin to block activin and BMP-type signaling in the embryo. Follistatin in mice is expressed in the primitive streak at gastrulation, and later in the developing hindbrain, somites, vibrissae, teeth, epidermis, and muscle. This chapter presents a study in which the homozygous mutant mice were recognizable at term by growth-retardation and their shiny and taut skin. The mutant mice failed to breathe and died within hours of birth. Musculoskeletal defects including cleft palate, delayed incisor development, hard palate anomalies, rib and lumbar vertebral defects, reduced mass of intercostals, and diaphragmatic muscles were found. Widespread defects probably result from altered regulation of several TGFβ-related proteins." @default.
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- W4252410855 date "1998-01-01" @default.
- W4252410855 modified "2023-09-27" @default.
- W4252410855 title "Follistatin" @default.
- W4252410855 doi "https://doi.org/10.1016/b978-012466044-1/50198-8" @default.
- W4252410855 hasPublicationYear "1998" @default.
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