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• W4253048106 abstract "Platelet activation and aggregation play important roles in ischemic event occurrences in high-risk patients with coronary artery disease (CAD). Dual antiplatelet therapy with aspirin and a P2Y12 receptor inhibitor is a key strategy to prevent major adverse cardiovascular events in CAD patients undergoing percutaneous coronary intervention (PCI) or those with acute coronary syndrome (ACS). Interindividual variability in response to aspirin and clopidogrel administration along with the influence of different polymorphisms on platelet reactivity has been demonstrated in healthy subjects or CAD patients. Reduced pharmacodynamic effect and reduced clinical efficacy of clopidogrel have been well documented in PCI or ACS patients carrying a loss-of-function allele of the CYP2C19 gene. In addition, accumulating data from numerous clinical studies underscore the importance of high on-treatment platelet reactivity (HPR) as a prognostic risk factor. However, the influence of HPR and/or genotype on clinical outcomes must be assessed in the context of the overall disease risk level (ACS vs. non-ACS, diabetes vs. non-diabetes, and old age), post-PCI time (early vs. late) and ethnicity. (Korean J Med 2013;85:1-9). Keywords: Clopidogrel; Platelet; Genotyping; Ischemic event; Bleeding 중심 단어: í´ë¡œí”¼ë„ê·¸ë ; 혈소판; Genotyping; 허혈; 출혈" @default.
• W4253048106 created "2022-05-12" @default.
• W4253048106 creator A5057103601 @default.
• W4253048106 date "2013-01-01" @default.
• W4253048106 modified "2023-09-23" @default.
• W4253048106 title "Clopidogrel Resistance" @default.
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• W4253048106 doi "https://doi.org/10.3904/kjm.2013.85.1.1" @default.
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