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• W4253259232 abstract "Acquired immune deficiency syndrome (AIDS) is a secondary immune deficiency caused by infection with a human retrovirus called human immunodeficiency virus (HIV). HIV infects and kills CD4+ T-helper cells. This causes a profound, irreversible immunosuppression that, without treatment, rapidly progresses from opportunistic infections to death in almost all infected individuals. A critical factor in the progression of HIV infection is the way that HIV manages to subvert or avoid immune defense mechanisms. These mechanisms will be presented in more detail throughout this chapter. The process of immune activation, which is critical for protection against infection, may, in the case of HIV infection, actually serve to activate expression of HIV and accentuate progression of inapparent infection to AIDS. The major problem with the clinical application of polymerase chain reaction (PCR) is the well-known high risk of false-positive results. Loss of delayed-type hypersensitivity (DTH) skin reactivity to standard test antigens is an independent predictor of progression to AIDS in persons with HIV infection. Theoretically, infection with a retrovirus such as HIV could be controlled by interfering with some stage of the life cycle of the virus. The most effective therapy is directed to inhibition of reverse transcriptase (RT) using dideoxynucleosides. A list of the drugs approved by the U.S. Food and Drug Administration for the treatment of AIDS is given in this chapter. A table provides a list of some of the putative immunogens and adjuvants. An example of combination therapy is highly active antiretroviral therapy." @default.
• W4253259232 created "2022-05-12" @default.
• W4253259232 date "2001-04-02" @default.
• W4253259232 modified "2023-10-14" @default.
• W4253259232 title "Immunology of AIDS" @default.
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• W4253259232 doi "https://doi.org/10.1128/9781555818012.ch19" @default.
• W4253259232 hasPublicationYear "2001" @default.
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