Matches in SemOpenAlex for { <https://semopenalex.org/work/W4281717678> ?p ?o ?g. }
- W4281717678 endingPage "17" @default.
- W4281717678 startingPage "1" @default.
- W4281717678 abstract "Microglial cells serve as molecular sensors of the brain that play a role in physiological and pathological conditions. Under normal physiology, microglia are primarily responsible for regulating central nervous system homeostasis through the phagocytic clearance of redundant protein aggregates, apoptotic cells, damaged neurons, and synapses. Furthermore, microglial cells can promote and mitigate amyloid β phagocytosis and tau phosphorylation. Dysregulation of the microglial programming alters cellular morphology, molecular signaling, and secretory inflammatory molecules that contribute to various neurodegenerative disorders especially Alzheimer's disease (AD). Furthermore, microglia are considered primary sources of inflammatory molecules and can induce or regulate a broad spectrum of cellular responses. Interestingly, in AD, microglia play a double-edged role in disease progression; for instance, the detrimental microglial effects increase in AD while microglial beneficiary mechanisms are jeopardized. Depending on the disease stages, microglial cells are expressed differently, which may open new avenues for AD therapy. However, the disease-related role of microglial cells and their receptors in the AD brain remain unclear. Therefore, this review represents the role of microglial cells and their involvement in AD pathogenesis." @default.
- W4281717678 created "2022-06-13" @default.
- W4281717678 creator A5002440225 @default.
- W4281717678 creator A5017053390 @default.
- W4281717678 creator A5025207218 @default.
- W4281717678 creator A5027539618 @default.
- W4281717678 creator A5037838755 @default.
- W4281717678 creator A5041691798 @default.
- W4281717678 creator A5046315023 @default.
- W4281717678 creator A5051669394 @default.
- W4281717678 creator A5087372145 @default.
- W4281717678 date "2022-06-02" @default.
- W4281717678 modified "2023-10-09" @default.
- W4281717678 title "Microglia in Alzheimer’s Disease: A Favorable Cellular Target to Ameliorate Alzheimer’s Pathogenesis" @default.
- W4281717678 cites W121687570 @default.
- W4281717678 cites W123631083 @default.
- W4281717678 cites W127741752 @default.
- W4281717678 cites W1493890929 @default.
- W4281717678 cites W1503511302 @default.
- W4281717678 cites W1517477926 @default.
- W4281717678 cites W1518371860 @default.
- W4281717678 cites W1558100964 @default.
- W4281717678 cites W1563590122 @default.
- W4281717678 cites W1581267597 @default.
- W4281717678 cites W1585245628 @default.
- W4281717678 cites W1597763516 @default.
- W4281717678 cites W1597844192 @default.
- W4281717678 cites W1607556721 @default.
- W4281717678 cites W1664596139 @default.
- W4281717678 cites W1775237831 @default.
- W4281717678 cites W1937826833 @default.
- W4281717678 cites W1963968275 @default.
- W4281717678 cites W1965395547 @default.
- W4281717678 cites W1970347605 @default.
- W4281717678 cites W1970626421 @default.
- W4281717678 cites W1971684738 @default.
- W4281717678 cites W1971963029 @default.
- W4281717678 cites W1972784541 @default.
- W4281717678 cites W1973838846 @default.
- W4281717678 cites W1974105677 @default.
- W4281717678 cites W1976625832 @default.
- W4281717678 cites W1978472910 @default.
- W4281717678 cites W1979755700 @default.
- W4281717678 cites W1979958331 @default.
- W4281717678 cites W1980543900 @default.
- W4281717678 cites W1981310043 @default.
- W4281717678 cites W1982161730 @default.
- W4281717678 cites W1984232954 @default.
- W4281717678 cites W1985060701 @default.
- W4281717678 cites W1985814840 @default.
- W4281717678 cites W1991763618 @default.
- W4281717678 cites W1991963544 @default.
- W4281717678 cites W1993748281 @default.
- W4281717678 cites W1996024251 @default.
- W4281717678 cites W1998079087 @default.
- W4281717678 cites W1999295089 @default.
- W4281717678 cites W2001285655 @default.
- W4281717678 cites W2001334299 @default.
- W4281717678 cites W2001618696 @default.
- W4281717678 cites W2002774469 @default.
- W4281717678 cites W2007197481 @default.
- W4281717678 cites W2008423294 @default.
- W4281717678 cites W2009110155 @default.
- W4281717678 cites W2011041653 @default.
- W4281717678 cites W2013100814 @default.
- W4281717678 cites W2014200126 @default.
- W4281717678 cites W2015426193 @default.
- W4281717678 cites W2018708829 @default.
- W4281717678 cites W2026060240 @default.
- W4281717678 cites W2030567293 @default.
- W4281717678 cites W2031002692 @default.
- W4281717678 cites W2031551713 @default.
- W4281717678 cites W2032694200 @default.
- W4281717678 cites W2033726284 @default.
- W4281717678 cites W2033906528 @default.
- W4281717678 cites W2034502915 @default.
- W4281717678 cites W2038048357 @default.
- W4281717678 cites W2038870006 @default.
- W4281717678 cites W2039313393 @default.
- W4281717678 cites W2041851050 @default.
- W4281717678 cites W2042895002 @default.
- W4281717678 cites W2045560271 @default.
- W4281717678 cites W2045720033 @default.
- W4281717678 cites W2045772297 @default.
- W4281717678 cites W2047658053 @default.
- W4281717678 cites W2050697792 @default.
- W4281717678 cites W2051539926 @default.
- W4281717678 cites W2051860681 @default.
- W4281717678 cites W2052742260 @default.
- W4281717678 cites W2054082659 @default.
- W4281717678 cites W2057249855 @default.
- W4281717678 cites W2061510546 @default.
- W4281717678 cites W2061715282 @default.
- W4281717678 cites W2064487089 @default.
- W4281717678 cites W2066344383 @default.
- W4281717678 cites W2067267561 @default.
- W4281717678 cites W2069245229 @default.
- W4281717678 cites W2069663822 @default.