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- W4281750672 endingPage "108887" @default.
- W4281750672 startingPage "108887" @default.
- W4281750672 abstract "Interleukin (IL)-33 is a member of the IL-1 cytokine family, which serves as both a cytokine and a nuclear factor. IL-33 is primarily expressed by keratinocytes, epithelial and endothelial cells, as well as human monocytes, Dendritic cells (DC), and astrocytes. IL-33 has been described as a modulator of inflammation and immune responses, which is mainly associated with the development of T helper 2 (Th2) immunological responses. Cytokines that control the T cells activation have been shown to have a role in the B-cell hyperactivity and production of pathogenic autoantibody isoforms that cause tissue dysfunction in autoimmune disorders. IL-33 levels have been shown to be elevated in a variety of autoimmune disorders, including Rheumatoid arthritis (RA), Systemic lupus erythematosus (SLE), type 1 diabetes (T1D), Multiple sclerosis (MS), and Inflammatory bowel diseases (IBD), suggesting that it may have a role in inducing autoimmunity and inflammatory damage. In addition to pathogenic functions in autoimmune disorders, cytokines may be a promising target for autoimmune disease treatment and follow-up. Therefore, the aim of this study is to review the possibility of using the IL-33/tumorigenicity 2 receptor (ST2) axis as a potential therapeutic target in the treatment of autoimmune disorders." @default.
- W4281750672 created "2022-06-13" @default.
- W4281750672 creator A5058272371 @default.
- W4281750672 date "2022-07-01" @default.
- W4281750672 modified "2023-10-18" @default.
- W4281750672 title "IL-33 in autoimmunity; possible therapeutic target" @default.
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