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• W4283713883 endingPage "1069" @default.
• W4283713883 startingPage "1050" @default.
• W4283713883 abstract "Sepsis-associated AKI is a life-threatening complication that is associated with high morbidity and mortality in patients who are critically ill. Although it is clear early supportive interventions in sepsis reduce mortality, it is less clear that they prevent or ameliorate sepsis-associated AKI. This is likely because specific mechanisms underlying AKI attributable to sepsis are not fully understood. Understanding these mechanisms will form the foundation for the development of strategies for early diagnosis and treatment of sepsis-associated AKI. Here, we summarize recent laboratory and clinical studies, focusing on critical factors in the pathophysiology of sepsis-associated AKI: microcirculatory dysfunction, inflammation, NOD-like receptor protein 3 inflammasome, microRNAs, extracellular vesicles, autophagy and efferocytosis, inflammatory reflex pathway, vitamin D, and metabolic reprogramming. Lastly, identifying these molecular targets and defining clinical subphenotypes will permit precision approaches in the prevention and treatment of sepsis-associated AKI." @default.
• W4283713883 created "2022-06-30" @default.
• W4283713883 creator A5043847672 @default.
• W4283713883 creator A5045984950 @default.
• W4283713883 creator A5091384892 @default.
• W4283713883 date "2022-07-01" @default.
• W4283713883 modified "2023-10-17" @default.
• W4283713883 title "The Pathophysiology of Sepsis-Associated AKI" @default.
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