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• W4285265055 abstract "Intracerebral hemorrhages due to traumatic brain injury are associated with dysregulation of the blood–brain barrier and can be critically involved in the acceleration of pathogenic processes leading to neurodegenerative diseases, including chronic traumatic encephalopathy and Alzheimer’s disease. After such hemorrhages, disruption of neurovascular coupling is frequently accompanied by vasogenic edema, abnormal ion concentrations in the brain parenchyma, improper neurotransmission, and post-traumatic ictogenesis. Brain bleeds are typically identified from susceptibility weighted imaging, which can be integrated with other forms of magnetic resonance imaging to evaluate the effects of hemorrhage upon the connectome and to evaluate patients’ risk of cognitive decline via patient-tailored mapping of white matter circuitry. While more frequent in older adults, males, diabetics, and hypertensives, traumatic hemorrhages may be difficult to distinguish from hemorrhages due to other causes, like cerebral amyloid angiopathy. Apolipoprotein and presenilin genotypes modulate risk of hemorrhage related to Alzheimer’s disease and traumatic brain injury and might forecast accelerated brain aging after injury, especially as reflected by epigenetic markers. This may reflect an interaction between genetic and traumatic risk factors for microbleeds and explain the relatively high frequency of hemorrhages in individuals at high risk for neurodegenerative diseases, as well as the finding that traumatic hemorrhages contributes to neurodegenerative disease risk in neurotrauma survivors." @default.
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• W4285265055 date "2022-01-01" @default.
• W4285265055 modified "2023-09-23" @default.
• W4285265055 title "Cerebral hemorrhages in traumatic brain injury" @default.
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• W4285265055 doi "https://doi.org/10.1016/b978-0-12-823347-4.00030-0" @default.
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