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- W4288045795 endingPage "310" @default.
- W4288045795 startingPage "300" @default.
- W4288045795 abstract "Endemic arsenism is a worldwide health problem. Chronic arsenic exposure results in cognitive dysfunction due to arsenic and its metabolites accumulating in hippocampus. As the cellular basis of cognition, synaptic plasticity is pivotal in arsenic-induced cognitive dysfunction. N-methyl-D-aspartate receptors (NMDARs) serve physiological functions in synaptic transmission. However, excessive NMDARs activity contributes to exitotoxicity and synaptic plasticity impairment. Here, we provide an overview of the mechanisms that NMDARs and their downstream signaling pathways mediate synaptic plasticity impairment due to arsenic exposure in hippocampal neurons, ways of arsenic exerting on NMDARs, as well as the potential therapeutic targets except for water improvement." @default.
- W4288045795 created "2022-07-27" @default.
- W4288045795 creator A5037677450 @default.
- W4288045795 creator A5088864773 @default.
- W4288045795 date "2022-08-01" @default.
- W4288045795 modified "2023-09-30" @default.
- W4288045795 title "N-Methyl-D-Aspartate Receptors Mediate Synaptic Plasticity Impairment of Hippocampal Neurons Due to Arsenic Exposure" @default.
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