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- W4288074603 endingPage "220060" @default.
- W4288074603 startingPage "220060" @default.
- W4288074603 abstract "Lung transplantation is the optimal treatment for selected patients with end-stage chronic lung diseases. However, chronic lung allograft dysfunction remains the leading obstacle to improved long-term outcomes. Traditionally, lung allograft rejection has been considered primarily as a manifestation of cellular immune responses. However, in reality, an array of complex, interacting and multifactorial mechanisms contribute to its emergence. Alloimmune-dependent mechanisms, including T-cell-mediated rejection and antibody-mediated rejection, as well as non-alloimmune injuries, have been implicated. Moreover, a role has emerged for autoimmune responses to lung self-antigens in the development of chronic graft injury. The aim of this review is to summarise the immune processes involved in the pathogenesis of chronic lung allograft dysfunction, with advanced insights into the role of innate immune pathways and crosstalk between innate and adaptive immunity, and to identify gaps in current knowledge." @default.
- W4288074603 created "2022-07-28" @default.
- W4288074603 creator A5004295201 @default.
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- W4288074603 creator A5020108764 @default.
- W4288074603 creator A5033618310 @default.
- W4288074603 creator A5077353591 @default.
- W4288074603 date "2022-07-27" @default.
- W4288074603 modified "2023-10-18" @default.
- W4288074603 title "Immune processes in the pathogenesis of chronic lung allograft dysfunction: identifying the missing pieces of the puzzle" @default.
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