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- W4292581714 endingPage "497" @default.
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- W4292581714 abstract "Accumulating evidence indicates that ataxia-telangiectasia mutated kinase is critical for maintaining cellular homeostasis and that it has both nuclear and cytoplasmic functions. However, the functions of ataxia-telangiectasia mutated that when lost lead to cerebellar degeneration are still unknown. In this review, we first describe the role of ataxia-telangiectasia mutated in cerebellar pathology. In addition to its canonical nuclear functions in DNA damage response circuits, ataxia-telangiectasia mutated functions in various cytoplasmic and mitochondrial processes that are critically important for cellular homeostasis. We discuss these functions with a focus on the role of ataxia-telangiectasia mutated in maintaining the homeostatic redox state. Finally, we describe the unique functions of ataxia-telangiectasia mutated in various types of neuronal and glial cells including cerebellar granule neurons, astrocytes, and microglial cells." @default.
- W4292581714 created "2022-08-22" @default.
- W4292581714 creator A5020543516 @default.
- W4292581714 creator A5071407320 @default.
- W4292581714 date "2023-01-01" @default.
- W4292581714 modified "2023-09-27" @default.
- W4292581714 title "Ataxia-telangiectasia mutated plays an important role in cerebellar integrity and functionality" @default.
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- W4292581714 doi "https://doi.org/10.4103/1673-5374.350194" @default.
- W4292581714 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/36018153" @default.
- W4292581714 hasPublicationYear "2023" @default.
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