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• W4293574530 abstract "There is now a consensus that mitochondria are important tumor drivers, sophisticated biological machines that can engender a panoply of key disease traits. How this happens, however, is still mostly elusive. The opinion presented here is that what cancer exploits are not the normal mitochondria of oxygenated and nutrient-replete tissues, but the unfit, damaged, and dysfunctional organelles generated by the hostile environment of tumor growth. These 'ghost' mitochondria survive quality control and thwart cell death to relay multiple comprehensive 'danger signals' of metabolic starvation, cellular stress, and reprogrammed gene expression. The result is a new, treacherous cellular phenotype, proliferatively quiescent but highly motile, that enables tumor cell escape from a threatening environment and colonization of distant, more favorable sites (metastasis)." @default.
• W4293574530 created "2022-08-30" @default.
• W4293574530 creator A5082346201 @default.
• W4293574530 date "2023-04-01" @default.
• W4293574530 modified "2023-09-29" @default.
• W4293574530 title "Mitochondria in cancer: clean windmills or stressed tinkerers?" @default.
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• W4293574530 doi "https://doi.org/10.1016/j.tcb.2022.08.001" @default.
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