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- W4293579172 abstract "Background and Aims : Myocardial ischemia reperfusion injury (MI-R) is the cellular damage or dysfunction of cardiac tissue caused by reperfusion induced oxidative stress and inflammation following ischemia. Malondialdehyde epitopes (MDA/MAA) are end-products of lipid peroxidation. MDA/MAA are enriched in the lesion site of patients that suffered a myocardial infarction and have the ability to increase apoptosis, inflammation and induce new ROS formation. Our aim is to further investigate and develop a 3D heart-on chip model to study the molecular mechanisms of MI-R and the role/formation of MDA/MAA in a physiological context." @default.
- W4293579172 created "2022-08-30" @default.
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- W4293579172 date "2022-08-01" @default.
- W4293579172 modified "2023-10-16" @default.
- W4293579172 title "Investigating the oxidative stress epitope malondialdehyde in myocardial ischemia reperfusion injury in a 2D and 3D heart-on-chip setting" @default.
- W4293579172 doi "https://doi.org/10.1016/j.atherosclerosis.2022.06.996" @default.
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