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• W4294032364 abstract "Vascular endothelial growth factor (VEGF) serves a critical role in vasculogenesis, angiogenesis, tumor, inflammatory angiogenesis and lymphangiogenesis. Since 2004, bevacizumab (Avastin), a humanized anti-VEGFA monoclonal antibody, has been approved for the treatment of non-small cell lung, breast, kidney and ovarian cancer in combination with standard chemotherapy. VEGF has been demonstrated to be important in the clinic as a therapeutic target in the anti-angiogenic approach to cancer therapy. The targeting of VEGF, together with immunotherapy, has been reported to be able to reverse the immunosuppressive effects of VEGF. A positive correlation between VEGF expression and the reduced survival rates of patients with cancer has also been demonstrated. Furthermore, increased VEGF expression can lead to immune suppression via the inhibition of dendritic cell maturation, the reduction of T-cell tumor infiltration and the promotion of inhibitory cell types in the tumor microenvironment." @default.
• W4294032364 created "2022-09-01" @default.
• W4294032364 creator A5054370212 @default.
• W4294032364 date "2022-09-01" @default.
• W4294032364 modified "2023-09-30" @default.
• W4294032364 title "Immunosuppressive effects of vascular endothelial growth factor (Review)" @default.
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• W4294032364 doi "https://doi.org/10.3892/ol.2022.13489" @default.
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