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- W4294114338 abstract "Diseases with T-helper cell subset imbalance involve multiple systems and organs. In addition to this, the pathogenesis of these diseases is always complex, and involves Th1, Th2, Th9, Th17, Th22, and Tfh cells. T-helper cell subset imbalance mediates immune responses to various pathogenic factors, by secreting specific cytokines. Although several studies have revealed the specific mechanisms of the occurrence and development of these diseases from different aspects, there is still a need for more comprehensive and in-depth studies that can compensate for the corresponding gaps in the diagnosis, targeted therapy, and prognosis of these diseases. N6-methyladenosine(m6A) modification is the most prevalent and abundant post-transcriptional modification in eukaryotic RNAs. In recent years, the critical role of m6A modification has been confirmed in multiple diseases with T-helper cell subset imbalance. m6A modification affects the immune cell development, inflammatory processes, biological behaviour of tumours, and immune response in these diseases. In this review, we focussed on how the enzymes involved in m6A modification, directly or indirectly, influence the pathogenesis and phenotype of various diseases with T-helper cell subset imbalance, and could therefore, serve as potential diagnostic markers and therapeutic targets for these diseases. In addition, this review also discusses the focus of future research in this area. Finally, we summarise the prospects of m6A modification in immunotherapy and chemotherapy." @default.
- W4294114338 created "2022-09-02" @default.
- W4294114338 creator A5018516457 @default.
- W4294114338 creator A5030564753 @default.
- W4294114338 creator A5049075415 @default.
- W4294114338 creator A5053413262 @default.
- W4294114338 creator A5056437521 @default.
- W4294114338 creator A5060389151 @default.
- W4294114338 creator A5082998428 @default.
- W4294114338 date "2022-11-01" @default.
- W4294114338 modified "2023-09-29" @default.
- W4294114338 title "Critical role of m6A modification in T-helper cell disorders" @default.
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