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- W4294771105 endingPage "2050" @default.
- W4294771105 startingPage "2039" @default.
- W4294771105 abstract "Hepatocellular carcinoma (HCC) is the most common liver malignancy that can be developed from hepatitis B and cirrhosis. Many pathophysiological alterations, including hepatitis B virus (HBV) DNA integration, oxidative stress, cytokine release, telomerase homeostasis, mitochondrial damage, epigenetic modification, and tumor microenvironment, are involved in the biological process from hepatitis B to cirrhosis and HCC. N6-methyladenosine (m6A), as an epitranscriptomic modification of RNAs, can regulate the stability, splicing, degradation, transcription, and translation of downstream target RNAs in HBV and liver cancer cells. m6A regulators (writers, erasers, and readers) play an important role in the pathogenesis of HBV-associated HCC by regulating cell proliferation, apoptosis, migration, autophagy, differentiation, inflammation, angiogenesis, and tumor microenvironment. This review summarizes the current progress of m6A methylation in the molecular mechanisms, biological functions, and potential clinical implications of HBV-associated HCC." @default.
- W4294771105 created "2022-09-06" @default.
- W4294771105 creator A5004992708 @default.
- W4294771105 creator A5017140872 @default.
- W4294771105 creator A5061599798 @default.
- W4294771105 creator A5064106247 @default.
- W4294771105 creator A5064466730 @default.
- W4294771105 creator A5076837264 @default.
- W4294771105 date "2022-09-20" @default.
- W4294771105 modified "2023-10-16" @default.
- W4294771105 title "Role of <scp>m6A</scp> RNA methylation in the development of hepatitis B virus‐associated hepatocellular carcinoma" @default.
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