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• W4298077473 abstract "As has been discussed, human basophil degranulation mediated by IgE or other stimuli leads to the release of histamine, sulfidopeptide leukotrienes, and other potent chemical mediators of inflammation. Basophil influx into sites of late-phase allergic reactions in the nose and skin has been demonstrated, and the release of their inflammatory mediators at these sites may be important in the pathogenesis of these responses. It is unknown whether a similar influx of basophils is present in late asthmatic responses occurring after specific antigen challenge in atopic asthmatics. As summarized, many inflammatory cells, including lymphocytes, macrophages, and neutrophils, release proteins that have potent histamine-releasing activity for basophils. Biochemical characterization of these HRFs will elucidate their relationship to known cytokines, and future research should define their role in stimulating release of basophil-derived chemical mediators that could play an important role in the mediation of the epithelial injury, mucus hypersecretion, and bronchial hyperreactivity that is characteristic of chronic asthma." @default.
• W4298077473 created "2022-10-01" @default.
• W4298077473 creator A5035302672 @default.
• W4298077473 date "1990-05-01" @default.
• W4298077473 modified "2023-09-26" @default.
• W4298077473 title "Basophils" @default.
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• W4298077473 doi "https://doi.org/10.1016/s0889-8561(22)00268-5" @default.
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