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- W4306864055 abstract "Inflammation is a major adverse outcome induced by inhaled particulate matter with a diameter of ≤ 2.5 µm (PM2.5), and a critical trigger of most PM2.5 exposure-associated diseases. However, the key molecular events regulating the PM2.5-induced airway inflammation are yet to be elucidated. Considering the critical role of circular RNAs (circRNAs) in regulating inflammation, we predicted 11 circRNAs that may be involved in the PM2.5-induced airway inflammation using three previously reported miRNAs through the starBase website. A novel circRNA circ_0008553 was identified to be responsible for the PM2.5-activated inflammatory response in human bronchial epithelial cells (16HBE) via inducing oxidative stress. Using a combinatorial model PM2.5 library, we found that the synergistic effect of the insoluble core and loaded Zn2+ ions at environmentally relevant concentrations was the major contributor to the upregulation of circ_0008553 and subsequent induction of oxidative stress and inflammation in response to PM2.5 exposures. Our findings provided new insight into the intervention of PM2.5-induced adverse outcomes." @default.
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- W4306864055 date "2022-10-01" @default.
- W4306864055 modified "2023-09-26" @default.
- W4306864055 title "REPORT OF SYSTEMIC EBV-POSITIVE T-CELL LYMPHOMA OF CHILDHOOD ASSOCIATED WITH XMEN DISEASE CAUSED BY A NOVEL MUTATION" @default.
- W4306864055 doi "https://doi.org/10.1016/s0145-2126(22)00283-1" @default.
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