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• W4306989438 endingPage "11" @default.
• W4306989438 startingPage "1" @default.
• W4306989438 abstract "Overview Chronic myeloid leukemia (CML) was the first malignancy where a unique chromosomal abnormality, the Philadelphia chromosome (Ph), was directly linked to it. The unraveling of the molecular and physiologic consequences of this abnormality, with a fusion gene ( BCR‐ABL1 ) translating into a chimeric protein with increased tyrosine kinase activity, led to the development of specific tyrosine kinase inhibitors (TKIs). The use of such agents as initial therapy has resulted in a dramatic change in the natural history of CML where patients properly managed can have a normal life expectancy. Several treatment options are also available for patients who may not have optimal response to their initial therapy. Current research goals are to improve the safety, efficacy, and wide availability of TKIs and to achieve a response such that patients can eventually discontinue therapy. With these approaches and ongoing research, the goal of eradication of chronic myeloid leukemia is ever closer." @default.
• W4306989438 created "2022-10-22" @default.
• W4306989438 creator A5015101485 @default.
• W4306989438 creator A5040941721 @default.
• W4306989438 creator A5084379902 @default.
• W4306989438 date "2022-10-21" @default.
• W4306989438 modified "2023-10-15" @default.
• W4306989438 title "Chronic Myeloid Leukemia" @default.
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