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- W4307942364 endingPage "13229" @default.
- W4307942364 startingPage "13229" @default.
- W4307942364 abstract "Sjögren’s syndrome (SS) is a systemic autoimmune rheumatic disorder characterized by the lymphocytic infiltration of exocrine glands and the production of autoantibodies to self-antigens. The involvement of the exocrine glands drives the pathognomonic manifestations of dry eyes (keratoconjunctivitis sicca) and dry mouth (xerostomia) that define sicca syndrome. To date, the molecular mechanisms mediating pathological salivary gland dysfunction in SS remain to be elucidated, despite extensive studies investigating the underlying cause of this disease, hampering the development of novel therapeutic strategies. Many researchers have identified a multifactorial pathogenesis of SS, including environmental, genetic, neuroendocrine, and immune factors. In this review, we explore the latest developments in understanding the molecular mechanisms involved in the pathogenesis of SS, which have attracted increasing interest in recent years." @default.
- W4307942364 created "2022-11-06" @default.
- W4307942364 creator A5012954143 @default.
- W4307942364 creator A5054370212 @default.
- W4307942364 creator A5067801834 @default.
- W4307942364 date "2022-10-30" @default.
- W4307942364 modified "2023-10-18" @default.
- W4307942364 title "Molecular Mechanisms Linking Inflammation to Autoimmunity in Sjögren’s Syndrome: Identification of New Targets" @default.
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- W4307942364 doi "https://doi.org/10.3390/ijms232113229" @default.
- W4307942364 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/36362017" @default.
- W4307942364 hasPublicationYear "2022" @default.
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