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- W4308029022 abstract "Rheumatoid arthritis (RA) occurs as the result of a complex interplay of environmental factors in a genetically susceptible individual. There is considerable evidence that the lungs may serve as an initial site of tolerance loss in the generation of RA-related autoimmunity, and several environmental inhalant exposures and lung diseases have been associated with RA risk. There is additional evidence that immune and microbial dysregulation of other mucosal sites, including the oral and gastrointestinal mucosa, may contribute to the development of RA. Epidemiologic evidence linking mucosal exposures to various environmental insults as risk determinants in RA will be reviewed." @default.
- W4308029022 created "2022-11-07" @default.
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- W4308029022 date "2022-11-01" @default.
- W4308029022 modified "2023-09-25" @default.
- W4308029022 title "Inhalant and Additional Mucosal-Related Environmental Risks for Rheumatoid Arthritis" @default.
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- W4308029022 doi "https://doi.org/10.1016/j.rdc.2022.06.002" @default.
- W4308029022 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/36332995" @default.
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