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- W4308208034 endingPage "293" @default.
- W4308208034 startingPage "280" @default.
- W4308208034 abstract "Neutrophil extracellular traps (NETs) evolved to protect the host against microbial infections and are formed by a web-like structure of DNA that is decorated with antimicrobial effectors. Due to their potent inflammatory functions, NETs also cause tissue damage and can favor and/or aggravate inflammatory diseases. This multipronged activity of NETs requires that the induction, release, and degradation of NETs are tightly regulated. Here we describe the key pathways that are intrinsic to neutrophils and regulate NETosis, and we review the most recent findings on how neutrophil extrinsic factors participate in the formation of NETs. In particular, we emphasize how bystander cells contribute to modifying the capacity of neutrophils to undergo NETosis. Finally, we discuss how these neutrophil extrinsic processes can be harnessed to protect the host against the excessive inflammation elicited by uncontrolled NET release." @default.
- W4308208034 created "2022-11-09" @default.
- W4308208034 creator A5050715964 @default.
- W4308208034 creator A5069622204 @default.
- W4308208034 date "2023-03-01" @default.
- W4308208034 modified "2023-10-17" @default.
- W4308208034 title "Neutrophil intrinsic and extrinsic regulation of NETosis in health and disease" @default.
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