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• W4308335828 startingPage "1637" @default.
• W4308335828 abstract "The endoplasmic reticulum (ER) is an integral organelle for maintaining protein homeostasis. Multiple factors can disrupt protein folding in the lumen of the ER, triggering ER stress and activating the unfolded protein response (UPR), which interrelates with various damage mechanisms, such as inflammation, apoptosis, and autophagy. Numerous studies have linked ER stress and UPR to the progression of chronic obstructive pulmonary disease (COPD). This review focuses on the mechanisms of other cellular processes triggered by UPR and summarizes drug intervention strategies targeting the UPR pathway in COPD to explore new therapeutic approaches and preventive measures for COPD." @default.
• W4308335828 created "2022-11-11" @default.
• W4308335828 creator A5035045235 @default.
• W4308335828 creator A5040553393 @default.
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• W4308335828 creator A5084029348 @default.
• W4308335828 date "2022-11-04" @default.
• W4308335828 modified "2023-10-13" @default.
• W4308335828 title "Endoplasmic Reticulum Stress in Chronic Obstructive Pulmonary Disease: Mechanisms and Future Perspectives" @default.
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