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- W4309358467 abstract "Jaundice, the yellow discoloration of skin and sclerae, results when the serum level of bilirubin, a pigmented compound, is elevated. Bilirubin is formed from the degradation of heme-containing compounds, particularly hemoglobin. Jaundice is not evident until the total serum bilirubin is at least 2–2.5 mg/dL in children out of the neonatal period. Excess of the pigment bilirubin may be caused by hemolysis, liver disease, or obstruction of the bile duct. Hyperbilirubinemia can be classified as conjugated (direct) or unconjugated (indirect), depending on the concentration in the serum. In the newborn, jaundice is a common condition due to the immature liver, although more significant underlying conditions must be considered. In evaluating the older child or adolescent with jaundice, it is important to determine whether the condition represents a hepatobiliary problem, a hematologic disorder, or a systemic illness. Coagulopathy, encephalopathy, or hypoglycemia may alert the clinician to hepatic failure, which mandates early assessment and intervention." @default.
- W4309358467 created "2022-11-26" @default.
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- W4309358467 date "2023-01-01" @default.
- W4309358467 modified "2023-10-14" @default.
- W4309358467 title "Jaundice" @default.
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- W4309358467 doi "https://doi.org/10.1016/b978-0-323-76174-1.00018-3" @default.
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