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- W4309553627 startingPage "114004" @default.
- W4309553627 abstract "When the production of reactive oxygen species (ROS) is overloaded surpassing the capacity of the reductive rheostat, mammalian cells undergo a series of oxidative damage termed oxidative stress (OS). This phenomenon is ubiquitously detected in many human pathological conditions. Wound healing program implicates continuous neovascularization, cell proliferation, and wound remodeling. Increasing evidence indicates that reactive oxygen species (ROS) have profound impacts on the wound healing process through regulating a series of the physiological and pathological program including inflammatory response, cell proliferation, angiogenesis, granulation as well as extracellular matrix formation. In most pathological wound healing processes, excessive ROS exerts a negative role on the wound healing process. Interestingly, the moderate increase of ROS levels is beneficial in killing bacteria at the wound site, which creates a sterile niche for revascularization. In this review, we discussed the physiological rhythms of wound healing and the role of ROS in this progress, aim to explore the potential manipulation of OS as a promising therapeutic avenue." @default.
- W4309553627 created "2022-11-28" @default.
- W4309553627 creator A5005071507 @default.
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- W4309553627 creator A5083454745 @default.
- W4309553627 creator A5088431767 @default.
- W4309553627 date "2023-01-01" @default.
- W4309553627 modified "2023-10-18" @default.
- W4309553627 title "The initiation of oxidative stress and therapeutic strategies in wound healing" @default.
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