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- W4310558241 endingPage "297" @default.
- W4310558241 startingPage "271" @default.
- W4310558241 abstract "Amphetamines represent a class of widely abused psychotropic drugs. Methamphetamine, amphetamine, and 3,4-methylenedioxymethamphetamine (“ecstasy”) represent well-recognized examples, but the use of natural, amphetamine-like compounds, including cathinone derivates, has been part of the human history for thousands of years. A variety of acute and long-term adverse effects have been disclosed in drug abusers, including neurotoxicity, in which a major role for mitochondria dysfunction has been depicted. At this level, perturbations in enzymes of tricarboxylic acid cycle, inhibition of electron transport chain components, and oxidative modifications in mitochondrial macromolecules represent well reported mechanisms underlying the neurotoxicity of these drugs. In this chapter, the toxic effects of amphetamines at the mitochondrial level are reviewed." @default.
- W4310558241 created "2022-12-12" @default.
- W4310558241 creator A5018784105 @default.
- W4310558241 creator A5060922570 @default.
- W4310558241 creator A5065222544 @default.
- W4310558241 date "2023-01-01" @default.
- W4310558241 modified "2023-10-02" @default.
- W4310558241 title "Mitochondrial disruption as a contributing factor for the neurotoxicity of amphetamines" @default.
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