Matches in SemOpenAlex for { <https://semopenalex.org/work/W4310583315> ?p ?o ?g. }
- W4310583315 endingPage "404" @default.
- W4310583315 startingPage "393" @default.
- W4310583315 abstract "Antiretroviral therapy (ART) reduces human immunodeficiency virus type 1 (HIV-1) infection, but selection of treatment-refractory variants remains a major challenge. HIV-1 encodes 16 canonical proteins, a small number of which are the singular targets of nearly all antiretrovirals developed to date. Cellular factors are increasingly being explored, which may present more therapeutic targets, more effectively target certain aspects of the viral replication cycle, and/or limit viral escape. Unlike most other positive-sense RNA viruses that encode at least one helicase, retroviruses are limited to the host repertoire. Accordingly, HIV-1 subverts DEAD-box helicase 3X (DDX3X) and numerous other cellular helicases of the Asp-Glu-x-Asp/His (DExD/H)-box family to service multiple aspects of its replication cycle. Here we review DDX3X and other DExD/H-box helicases in HIV-1 replication and their inhibition." @default.
- W4310583315 created "2022-12-12" @default.
- W4310583315 creator A5019562426 @default.
- W4310583315 creator A5051823046 @default.
- W4310583315 creator A5091208578 @default.
- W4310583315 date "2023-04-01" @default.
- W4310583315 modified "2023-09-30" @default.
- W4310583315 title "DExD/H-box helicases in HIV-1 replication and their inhibition" @default.
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