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- W4311348216 abstract "Subarachnoid haemorrhage (SAH) is a common cerebrovascular disease with high disability and mortality rates worldwide. The pathophysiological mechanisms involved in an aneurysm rupture in SAH are complex and can be divided into early brain injury and delayed brain injury. The initial mechanical insult results in brain tissue and vascular disruption with hemorrhages and neuronal necrosis. Following this, the secondary injury results in diffused cerebral damage in the peri-core area. However, the molecular mechanisms of neuronal death following an aneurysmal SAH are complex and currently unclear. Furthermore, multiple cell death pathways are stimulated during the pathogenesis of brain damage. Notably, particular attention should be devoted to necrosis, apoptosis, autophagy, necroptosis, pyroptosis and ferroptosis. Thus, this review discussed the mechanism of neuronal death and its influence on brain injury after SAH." @default.
- W4311348216 created "2022-12-25" @default.
- W4311348216 creator A5000631538 @default.
- W4311348216 creator A5002585720 @default.
- W4311348216 creator A5011376657 @default.
- W4311348216 creator A5034518696 @default.
- W4311348216 creator A5055725882 @default.
- W4311348216 date "2022-12-13" @default.
- W4311348216 modified "2023-10-13" @default.
- W4311348216 title "Molecular mechanisms of neuronal death in brain injury after subarachnoid hemorrhage" @default.
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