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• W4313175099 endingPage "2597" @default.
• W4313175099 startingPage "2585" @default.
• W4313175099 abstract "Gastric cancer (GC) or stomach cancer is a form of malignancy originating from the gastric epithelium. GC accounts for approximately 5.6% of all new cancers diagnosed each year and is the fifth most common reason for cancer-related fatality globally. Reactive oxygen species (ROS) are essential components engaged in the biological development of GC. In a tumor microenvironment, ROS is responsible for initiating angiogenesis, metastasis, and cancer cell survival at varied concentrations. It is important for either augmenting tumorigenesis or causing apoptosis of tumor cells. ROS nitric oxide (NO) and malondialdehyde (MDA) act as GC’s primary causative agent. Consumption of alcohol and the presence of stomach bacterium Helicobacter pylori are linked with ROS formation in GC. ROS directs the damage of gastric tissues by attacking the DNA or disrupting cellular signaling. Bone marrow-derived cells (BMDCs) or progenitor cells are proposed as gastric cancer stem cells (GCSCs) candidates. BMDCs contribute to GC by migrating and repopulating the gastric mucosa during infections. Understanding the role of GCSC in the tumor microenvironment is crucial for GC treatment. The emergence of novel candidates targeting the crucial mechanisms and molecules associated with the development of GCSCs and the encompassing environment may provide new opportunities for the comprehensive treatment of GC." @default.
• W4313175099 created "2023-01-06" @default.
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• W4313175099 date "2022-01-01" @default.
• W4313175099 modified "2023-09-26" @default.
• W4313175099 title "Current Insights to Therapeutic Targets of ROS-Induced Gastric Cancer Stem Cells" @default.
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• W4313175099 doi "https://doi.org/10.1007/978-981-16-5422-0_255" @default.
• W4313175099 hasPublicationYear "2022" @default.
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