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- W4313236551 endingPage "275" @default.
- W4313236551 startingPage "261" @default.
- W4313236551 abstract "Myocardial fibrosis is the heart's common healing response to injury. While initially seeking to optimize the strength of diseased tissue, fibrosis can become maladaptive, producing stiff poorly functioning and pro-arrhythmic myocardium. Different patterns of fibrosis are associated with different myocardial disease states, but the presence and quantity of fibrosis largely confer adverse prognosis. Current imaging techniques can assess the extent and pattern of myocardial scarring, but lack specificity and detect the presence of established fibrosis when the window to modify this process may have ended. For the first time, novel molecular imaging methods, including gallium-68 (68Ga)-fibroblast activation protein inhibitor positron emission tomography (68Ga-FAPI PET), may permit highly specific imaging of fibrosis activity. These approaches may facilitate earlier fibrosis detection, differentiation of active vs. end-stage disease, and assessment of both disease progression and treatment-response thereby improving patient care and clinical outcomes." @default.
- W4313236551 created "2023-01-06" @default.
- W4313236551 creator A5000133551 @default.
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- W4313236551 creator A5074252129 @default.
- W4313236551 creator A5080877411 @default.
- W4313236551 creator A5082758671 @default.
- W4313236551 date "2022-12-28" @default.
- W4313236551 modified "2023-10-17" @default.
- W4313236551 title "Emerging molecular imaging targets and tools for myocardial fibrosis detection" @default.
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