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• W4313333746 endingPage "1593" @default.
• W4313333746 startingPage "1589" @default.
• W4313333746 abstract "Abstract NK cells induce MHC class II molecules on the surface of allogeneic endothelial cells in an adhesion-dependent, IFN-γ-independent manner. Here, we demonstrate that NK cells induce HLA-DR on the surface of a mutant cell line that is defective in IFN-γ-induced MHC class II expression. RNA analysis in these cells and in a cell line that is defective in class II transactivator (CIITA) demonstrates that NK cell-induced HLA-DRα mRNA expression is also CIITA-independent. The Janus kinase-1-deficient cell line U4A expresses HLA-DRα mRNA in response to NK cell activation, and HLA-DRα promoter constructs transfected into these cells are induced by NK cells but not IFN-γ. These data indicate that the IFN-γ-independent component of the target cell HLA-DR expression induced by lymphocyte adhesion uses a signaling pathway that is distinct from the IFN-γ-dependent mechanism and also suggest that CIITA is not required." @default.
• W4313333746 created "2023-01-06" @default.
• W4313333746 creator A5004490478 @default.
• W4313333746 creator A5051385259 @default.
• W4313333746 creator A5082125129 @default.
• W4313333746 date "1998-08-15" @default.
• W4313333746 modified "2023-10-18" @default.
• W4313333746 title "Cutting Edge: Class II Transactivator-Independent Endothelial Cell MHC Class II Gene Activation Induced by Lymphocyte Adhesion" @default.
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• W4313333746 doi "https://doi.org/10.4049/jimmunol.161.4.1589" @default.
• W4313333746 hasPublicationYear "1998" @default.
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