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- W4313343196 abstract "With the global rise in incidences of ulcerative colitis and various dysbiosis in the gut microbiota, there has been a growing demand to understand the development and cause behind this disease. Numerous findings have been predicted that gut microbiome is involved in the development of the ulcerative colitis and could also delay the healing process. Studies suggest that the penetration of gut bacteria inside the intestinal wall cause the release of interferon gamma, tumor necrotic factors, interleukins-1 and 6 (IFN-γ, TNF-α, IL-1, and IL-6) that produce reactive oxygen species which further damages the intestinal lining and at the top delays the healing process. The major role is played by the regulatory T cells (Tregs) and the formation of interleukin-10 (IL-10Interleukin (IL)-10 (IL-10)) via GPR43. The Treg cells are stimulated by IL-10Interleukin (IL)-10 (IL-10) that causes activation of macrophages. Many pro-inflammatory mediators such as Th-1 and Th-17 are produced in response to invasive gut microorganisms (e.g., TNF-α, IFN-γ) by enhancing the transcription of significant genes. This chapter focuses on all the alternative pathophysiology and pathogenesis and management related to ulcerative colitis." @default.
- W4313343196 created "2023-01-06" @default.
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- W4313343196 date "2022-01-01" @default.
- W4313343196 modified "2023-09-27" @default.
- W4313343196 title "Microorganisms in Pathogenesis and Management of Ulcerative Colitis (UC)" @default.
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- W4313343196 doi "https://doi.org/10.1007/978-981-19-4800-8_12" @default.
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